Adult-onset obesity is the condition where individuals gain excessive weight after reaching adulthood, typically due to lifestyle factors. It can significantly affect neurological health through various metabolic and inflammatory pathways.
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Adult-onset obesity increases the risk of neurodegenerative diseases such as Alzheimer's and Parkinson's.
Chronic inflammation associated with adult-onset obesity can lead to cognitive decline.
Obesity in adulthood is linked to reduced levels of brain-derived neurotrophic factor (BDNF), impacting neuronal health.
Excessive weight gain in adults can impair the blood-brain barrier, making the brain more vulnerable to toxins.
Lifestyle changes including diet and exercise are critical for managing adult-onset obesity and its neurological impacts.
Review Questions
How does adult-onset obesity contribute to the risk of neurodegenerative diseases?
What role does chronic inflammation play in cognitive decline associated with adult-onset obesity?
Why are lifestyle changes important for managing the neurological impacts of adult-onset obesity?
Related terms
Neuroinflammation: Inflammation of nervous tissue, often linked to neurodegenerative diseases and affected by metabolic conditions like obesity.
Brain-Derived Neurotrophic Factor (BDNF): A protein crucial for neuron survival and plasticity; its levels can be negatively affected by obesity.
Blood-Brain Barrier: A selective barrier that protects the brain from harmful substances; its integrity can be compromised by obesity-related metabolic issues.